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    Canine Tick-Borne Encephalitis: Clinical Features, Survival Rate and Neurological Sequelae: A Retrospective Study of 54 Cases (–)

    Introduction

    Tick-borne encephalitis (TBE) is a viral disease, endemic in Europe, causing mainly nonspecific febrile illness followed by a remission phase in people. After this initial phase the disease may affect the nervous system in up to 50% of the cases. In approximately 30% to 80% of recovered patients, long-term neurological sequelae are observed (1&#x;4). The disease is caused by the European subtype of Flavivirus (TBEV), a single positive stranded RNA virus, that infects dogs, horses, domestic and wild ruminants, rodents, wild boars, and humans. In Eurasia, TBE has not only been associated with this subtype but also with the Siberian and the Far-Eastern subtypes that tends to show a mono-phasic pattern (1, 2). The TBEV is mainly inoculated by Ixodes spp. (4, 5) and Dermacentor spp. (6) ticks in Europe. The prevalence of TBEV within the tick population ranges from to 5% in the European countries (7, 8). In Switzerland, the prevalence is around % even in previously considered safe regions such as the southern site of the Alps (8). Epidemi

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    Tick-borne encephalitis virus in dogs - is this an issue?

    • Review
    • Open access
    • Published:

    Parasites & Vectorsvolume 4, Article number: 59 () Cite this article

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    Abstract

    The last review on Tick-borne encephalitis (TBE) in dogs was published almost ten years ago. Since then, this zoonotic tick-borne arbovirus has been geographically spreading and emerging in many regions in Eurasia and continues to do so. Dogs become readily infected with TBE virus but they are accidental hosts not capable to further spread the virus. They seroconvert upon infection but they seem to be much more resistant to the clinical disease than humans. Apart from their use as sentinels in endemic areas, however, an increasing number of case reports appeared during the last decade thus mirroring the rising public health concerns. Owing to the increased mobility of people travelling to endemic areas with their companion dogs, this consequently leads to problems in recognizing and diagnosing this severe infection in a yet non-endemic area, simply because the veterinarians are not considering TBE. This situation warrants an update on the epi

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    Risk för TBE-smitta inom opastöriserad mjölk

    TBE existerar en från de allvarligaste fästingburna sjukdomarna hos människor i Europa. Sjukdomen orsakas av TBE-virus, som främst överförs via fästingbett, dock även via konsumtion från opastöriserad mejeriprodukt eller opastöriserade mjölkprodukter såsom ost samt yoghurt. Nötkreatur, får samt getter förmå bli infekterade utan för att uppvisa symtom, men utsöndra viruset inom mjölken. Idag finns inga kända fall av livsmedelssmitta i landet, men sjukdomsutbrott och smittspridning förekommer inom andra delar av Europa där opastöriserade mjölkprodukter konsumeras i större utsträckning. till att förhindra smittspridning är kapabel mjölken pastöriseras.

    Kartläggning av riskområden för TBE

    Syftet med studien var för att identifiera samt kartlägga riskområden för TBE i land genom för att undersöka TBE-virusantikroppar i tankmjölksprover från mjölkbesättningar. De deltagande producenterna svarade på problem att diskutera om gårdens mjölkproduktion, pastörisering, fästingprofylax, fästingburna sjukdomar samt TBE-vaccination. oss analyserade mjölkproverna med ett ELISA-metod.

    Användbar teknik för övervakning av TBE

    TBE-virusantikroppar påvisades vid 20 gårdar. På dessa gårdar blev tagna nya tankm